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dc.contributor.authorLyman Gingerich, Jamie S.
dc.contributor.authorMeyer, Samantha
dc.date.accessioned2017-12-08T18:23:27Z
dc.date.available2017-12-08T18:23:27Z
dc.date.issued2017-12-08T18:23:27Z
dc.identifier.urihttp://digital.library.wisc.edu/1793/77511
dc.descriptionColor poster with text, graphs, charts, and images.en
dc.description.abstractThe focus of this project is to identify the downstream effects of mutations in the causative genes (PKD1 and PKD2) of polycystic kidney disease (ADPKD). ADPKD is responsible for 5% of all endstage renal disease and is characterized by large, fluid-filled cysts forming in kidney tubules and collecting ducts that disrupt the normal functioning of the kidneys. It is currently thought that primary cilia (1) and different signaling pathways may play a major role in the progression of ADPKD.en
dc.description.sponsorshipUniversity of Wisconsin--Eau Claire Office of Research and Sponsored Programsen
dc.language.isoen_USen
dc.relation.ispartofseriesUSGZE AS589;
dc.subjectC. elegansen
dc.subjectZebrafishen
dc.subjectPolycystic kidney diseaseen
dc.subjectPostersen
dc.titleZebrafish, C. elegans and Human Polycystic Kidney Diseaseen
dc.typePresentationen


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  • Student Research Day
    Posters of collaborative student/faculty research presented at Student Research Day

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